SchizophreniaThe Theorem: A Breakthrough Discovery in Schizophrenia!
Schizophrenia, the granddaddy of all behavioral disorders. Since the early 20th century, and perhaps before, when Dr. Kurt Schneider first began defining symptomology to this extremely complex disorder, the debate and disagreements have never stopped. Even today you would be hard pressed to find two psychiatrists who do not differ on opinions of symptomology.
When Arone made the discovery that would lead to The Theorem in 1994, schizophrenia was not included in the model. Autism, sleep, disorders of sleep, anorexia, OCD, panic disorder, and the rest were all discovered first. But his concept of one key to unlock all the doors of the disorders and behaviors would certainly be at risk if he could not at least make an assault on what was the biggest peak in psychiatric nomenclature. So he attempted, and did not summit. The result is the section you see in Chapter 13. It is good, interesting and is not contradictory to the rest of the theory. But it is not likely the correct model.
Fast forward almost 10 years later. It was after the book was accepted for publication, Arone was sifting through the research once again in preparation for the neural notes section. He realized the answer was there in front of him the entire time, prompting a new discovery and a lengthy delay. Too late to break up the cohesiveness of the book, the superior schizophrenia model discovery was forced into the Neural Notes Appendix. Did he get it right the second time? It is very possible, but the value is the perspective gained by any researcher; how the cohesiveness of the two cycles of intrauterine development generate both negative and positive symptoms.
While almost impossible to summarize here, essentially this fetus realizes accelerated memory and periods of primitive anticipatory learning in transitory states throughout the pregnancy. This is due to less than optimal intrauterine conditions caused from both genetic and environmental factors; such as influenza and immunological irregularities the mother realizes. The negative symptoms reflect the enhanced memory and lack of conformity to the First Fear Cycle of Development. This cycle is where the vast majority of motivational and reproductive aspects of the model are experienced. The positive symptoms of the disorder then are due to this lack of fetal conformity during the dopamine enriched Euphoric Cycle, of much lower importance developmentally. It makes sense when you realize that those with negative symptoms typically experience an earlier, during prime reproductive years, onset and a darker outlook. While those with positive symptoms historically have a better outlook when treated with antipsychotics. Though many exhibit mixed symptoms.
One of the major problems with schizophrenia is that this fetus realized a large amount of time conforming to the behavioral model the same as the typical fetus did. So you have essentially a layered consciousness, with dominance of either conflicting models influenced by a wide variety of factors including age and external events the sufferer realizes.
These chapters are simply a must read for anyone who deals with schizophrenia on a personal or a professional level. From psychiatrists, psychologists to anyone in the field of pharmacological research; there will be a day when this be your primer, skip it and the field could skip by you.